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Pain Lecture_Surgery Term I

Across
Accumulation of fluid in the lungs, can occur post-operatively 2/2 not ambulating
Not well localized, constant or intermittent mechanism of pain, i.e. GI obstruction
Conversion of noxious stimuli into electrical energy
Act on mu-receptors in brain/spinal cord, cause euphoria, sedation, C, and respiratory depression
Part of the Pain Modulating Network which interprets ascending nociceptive information
Leads to increased morbidity, poor QOL, delayed recovery, dependency on opioid
Condition of breathing at an irregularly increased rate
Increased risk of bleeding
Painful stimulus
CV response to uncontrolled pain
Pain that is well characterized, typically self-limited
Hypomotility of the gastrointestinal tract in absence of mechanical bowel obstruction
Well localized ad constant mechanism of pain, i.e. laceration
Down
Assessment of infant behavior concerning pain
Painful stimuli ascends the spinal cord to the higher cortical structures of the brain
Postoperative pain that may be treated with TENS
Demonstrates analgesic effect, undergoes extensive first-pass hepatic metabolism
Action of urinating, may be affected by anesthesia
Momentary shock-like pain over eye or in temporal or occipital regions
May be increased in response to uncontrolled pain, may be accompanied with dyspnea
Pain that persists greater than 3-6 months
Anesthetic, useful in delineating pain mechanisms, interrupt nociceptive activity. i.e. epidural
Component of the FLACC Scale