What is the likely ethnic background of a person with a haplotype of ce?
Patients with M pneumoniae infections are associated with what specificity of i?
My antibodies are sensitive to enzyme treatment.
Does not bind
complement.
AHG at 37C.
IgG.
Shows dosage.
Glycoprotein on the red cell membrane that
serves as RBC receptor for chemokines.
Expressed on cord blood cells.
I am a blood group system that is passively adsorbed onto RBC membranes from glycolipids present in plasma. (I am the undercover agent of blood group antigens).
I can cause problems in pregnancy b/c I decline dramatically, making the woman transiently have a neg phenotype, which can produce my antibodies. After delivery, her antigens return and the antibodies will disappear.
I am enhanced by ficin.
Neutralization is effective.
I am a blood group system associated with the resistance to P. vivax malaria?
Most weak D occurs in what ethnicity?
DCe. HOW WOULD THIS HAPLOTYPE BE WRITTEN IN WIENER NOTATION?
I am a rare, autosomal recessive phenotype that lacks all ABH antigens.
Down
If your patient was O+ and inherited FUT3 AND FUT2 genes, what would most likely be their ethnicity?
I am a blood group system that is not intrinsic to RBCs, I attach at the C4 component of complement. My antigens are mostly destroyed ficin and papain but are resistant to treatment with DTT. Neutralization is often used to identify my antibodies.
What ethnicity is most associated with Del?
I am a blood group system that consists of 32 different antigens. I am only found on RBCs. I am well developed at birth so HDFN is a concern.
My system is hard to detect and are a common cause of delayed HTRs.
What is the most common partial D type in people of European descent?
KEL 6 is almost completely confined to people of what ethnicity?
I am a cold-reacting saline agglutinin. I do not bind complement but can be destroyed by ficin. I often show dosage and can fall below detectable levels.
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