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Medications

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Across
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Opioid analgesic; Bind to opiate receptors in the CNS. Alter the perception of and response to painful stimuli while producing generalized CNS depression
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Antiplatelet; inhibits platelet aggregation by irreversibly inhibiting the binding of ATP to platelet receptors
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Antiarrhythmic; increases the force of myocardial contraction
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Diuretic; causes loss of sodium bicarbonate & calcium while saving potassium & hydrogen ions by antagonizing aldosterone
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Antianxiety; depresses the CNS
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Diuretic; (Lasix) Inhibits the reabsorption of sodium and chloride from the loop of Henle and distal renal tubule
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Hormones; replacement of or supplementation to endogenous thyroid hormones
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Antacid; mineral and electrolyte replacements and suspensions
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Antidepressant; selectively inhibits the reuptake of serotonin in the CNS
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Antihypertensive; ACE inhibitors block the conversion of angiotensin I to the vasoconstrictor angiotensin II
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Antidiabetic; Lowers blood glucose by stimulating glucose uptake in skeletal muscle and fat, inhibiting hepatic glucose production
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Anticoagulant; Acts as a selective, reversible site inhibitor of factor Xa, inhibiting both free and bound factor
Down
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Antipyretics, Non-opioid analgesics; produce analgesia and reduce inflammation and fever by inhibiting the production of prostaglandins. Decreases platelet aggregation
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Analgesic; decreases pain receptors
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Antiulcer; Inhibits the action of histamine at the H2 -receptor site located primarily in gastric parietal cells, resulting in inhibition of gastric acid secretion
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Analgesic adjuncts, anticonvulsants, mood stabilizers; Mechanism of action is not known. May affect transport of amino acids across and stabilize neuronal membranes
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Antiulcer; inhibits the action of histamine at the H2-receptor site located primarily in gastric parietal cells, resulting in inhibition of gastric acid secretion
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Antipyretics, Non-opioid analgesics; inhibits synthesis of prostaglandins that may serve as mediators of pain and fever, primarily in the CNS
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Lipid lowering; Inhibits 3-hydroxy-3-methylglutaryl-coenzyme A (HMG-CoA) reductase, an enzyme which is responsible for catalyzing an early step in the synthesis of cholesterol
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Antihypertensive; (Demadex) inhibits the reabsorption of sodium and chloride from the loop of Henle and distal renal tubule
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Antianginals, antihypertensives; blocks stimulation of beta1 (myocardial)-adrenergic receptors
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Lipid lowering; proposed mechanisms include ↑ activity of liver enzymes, ↓ hepatic lipogenesis, ↑ lipoprotein lipase activity and ↓ TG synthesis