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Drug Project - Action

Teacher: Keith Sautel
Across
A competitive nondepolarizing drug that competes with acetylcholine for receptor sites
Blocks pain impulses in CNS
Competes with histamine for H-1 receptor sites
Binds to opiate receptors in the CNS
Decreases inflammation by suppressing migration of Leukocytes and fibroblast
Prevents muscles from contracting by prolonging time during which the receptors at the neuromuscular junction cannot respond to ACh
Promotes reabsorption of water by action on renal tubular epithelium
Prevents nausea/vomiting by blocking serotonin peripherally
Rapidly increases blood glucose
Depresses pain impulse transmission at the spinal cord level
Dilates bronchial smooth muscle
Depresses the activity and reactivity of the brain stem reticular formation.
Down
Causes increased cardiac output; acts on B-1 and Alpha receptors
Stimulates Alpha & Beta adrenergic receptor causing increased levels of cAMP
CNS depressant causing sedative
Acts on the hepatocytes to stimulate release of glucose
Relaxes Vascular Smooth muscles, causes vasodilation
Slows conduction through AV node
Inhibits Calcium ion influx across cell membrane during cardiac depolarization
Prolongs duration of action potential and effective refractory period
Competes with opioids at oopiate receptor sites
Inhibits interaction of ACh at receptor sites on Bronchial Smooth muscle